1.
São Paulo; Guanabara Koogan; 12. ed; 2013. 552 p.
Monography
in Portuguese
| LILACS, ColecionaSUS
| ID: biblio-941481
2.
São Paulo; Guanabara Koogan; 12. ed; 2013. 552 p.
Monography
in Portuguese
| LILACS
| ID: lil-766468
3.
Rio de Janeiro; Guanabara Koogan; 2012. 489 p. ilus.
Monography
in Portuguese
| LILACS, EMS-Acervo
| ID: lil-655161
4.
São Paulo; Atheneu; 5 ed; 1999. 294 p. ilus, tab.
Monography
in Portuguese
| LILACS, HSPM-Acervo
| ID: lil-683692
5.
San Diego; Academic Press; 2nd; 1998. 145 p. ilus.
Monography
in English
| LILACS, ColecionaSUS
| ID: biblio-939139
6.
Rio de Janeiro; ATHENEU; 5 ed; 1989. 294 p. graf, ilus.(Biomédica: textos para a universidade).
Monography
in Portuguese
| LILACS, AHM-Acervo, TATUAPE-Acervo
| ID: lil-646271
7.
Mem. Inst. Oswaldo Cruz
;
82(supl.2): 105-109, 1987. tab, graf
Article
in English
| LILACS
| ID: lil-623770
ABSTRACT
1. Autoimmunity in deisease is driven by autoantigen; 2. Cell surface molecules may stimulate autoreactive T-helpers if call II MHC is expressed; special factors may predispose to the ease of class II induction; 3. Soluble autoantigens may be focussed by primed B-cells and processed for presentation to T-cell; 4. autoantigenicity may be influenced by metabolic events: (a) Poorly iodinated thyroglobulin does not induce thyroiditis; (b) IgG rheumatoid arthritis has galactose deficient Fc oligosaccharides. Glycosylation defects may prove to have wide implications.